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Endogenous Modulation of Pain - the Tolerance to Stretch
This study investigates the role of two endogenous inhibitory mechanisms; exercise-induced hypoalgesia (EIH) and a conditioning painful stimulus (CPM) on passive joint range of motion, passive resistive torque and pain sensitivity. The study is a randomized, repeated-measures cross-over study.
The increase in range of motion following stretching is related to changes in the tolerance to stretch. The mechanistic relationships behind these changes in subject sensation are however unknown but could be related to endogenous modulation of pain.
Age
18 - 45 years
Sex
MALE
Healthy Volunteers
Yes
University College of Northern Denmark
Aalborg, Denmark
Start Date
October 1, 2017
Primary Completion Date
December 30, 2017
Completion Date
December 30, 2017
Last Updated
March 26, 2019
19
ACTUAL participants
Exercise induced hypoalgesia (EIH)
OTHER
Static stretch (SS)
OTHER
Rest
OTHER
Conditioned pain modulation (CPM)
OTHER
Lead Sponsor
University College of Northern Denmark
Data Source & Attribution
This clinical trial information is sourced from ClinicalTrials.gov, a service of the U.S. National Institutes of Health.
Modifications: This data has been reformatted for display purposes. Eligibility criteria have been parsed into inclusion/exclusion sections. Location data has been geocoded to enable distance-based search. For the authoritative and most current information, please visit ClinicalTrials.gov.
Neither the United States Government nor Clareo Health make any warranties regarding the data. Check ClinicalTrials.gov frequently for updates.
View ClinicalTrials.gov Terms and ConditionsNCT07178288